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. Author manuscript; available in PMC: 2022 Feb 3.
Published in final edited form as: Neuron. 2020 Dec 8;109(3):473–487.e5. doi: 10.1016/j.neuron.2020.11.012

Figure 6: Overexpression of Wlds, or increasing the level of dNmnat, is sufficient to rescue axon transport defects after nerve injury.

Figure 6:

(A, B) Severed and intact axons expressing either WldS or dNmnat showed no changes in autophagosomes and synaptic vesicles (SV) transport after axonal injury 3hpa compared to that in uninjured wings. Two-way ANOVA with Sidak multiple comparisons test. (ns = not significant, *p < 0.05, **p < 0.01, ***p < 0.001, ****p < 0.0001, n = 10 axons of each, Error bar = S.E.M.). (C) Severed and intact axons of hiwΔN mutant clones showed comparable percentage of axon transport after axonal injury 3hpa with that in uninjured wings. Two-way ANOVA with Sidak multiple comparisons test. (ns = not significant, *p < 0.05, ****p < 0.0001, n = 10 axons of each, Error bar = S.E.M.)