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. 2021 Feb 5;7:16. doi: 10.1038/s41522-021-00184-w

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A Timeline of two in vitro chemostat models that were used to simulate primary CDI and recurrence after vancomycin treatment (black) and vancomycin treatment followed by FMT instillation (green). Luminal C. difficile recoveries from the recurrence model (B) and from FMT model (C). Both figures show the total viable counts (red lines), spores (blue lines) and period of toxin detection (black arrows). Results are shown as mean log₁₀ cfu/mL from two biological replicates, and three technical replicates from each. Error bars represent the standard deviation.