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. 2020 Sep 4;29(2):262–270. doi: 10.1038/s41431-020-00719-3

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A total of 1700 in-house individuals were screened for suspected hereditary FSGS. Decision to include patients was based on prioritization criteria, which led to the inclusion of 19 index patients. In addition, five samples were sent in for molecular analysis. A total of 29% of the cases could be solved. A total of 68% of the patients did not show any clear disease-causing variant.