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. 2021 Feb 8;20:31. doi: 10.1186/s12943-021-01324-8

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Clinical associations of m⁶A-modified TE lncRNAs across cancer types. a Heat map showing the number of TE lncRNA, m⁶A-modified TE lncRNAs, protective and risk lncRNAs, up- and down-regulated lncRNAs in each cancer. Cancer types were clustered together based on the overlap of TE lncRNAs. b Boxplots showing the expression of F11-AS1 in HCC patients and normal samples. c Kaplan-Meier plot for overall survival of HCC patients stratified by expression of F11-AS1. d Boxplots showing the expression of LINC01018 in HCC patients and normal samples. e Kaplan-Meier plot for overall survival of HCC patients stratified by expression of LINC01018. f Kaplan-Meier plot for overall survival of LGG patients stratified by expression of MIR325HG. g Kaplan-Meier plot for overall survival of GBM patients stratified by expression of MIR325HG