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. Author manuscript; available in PMC: 2021 Aug 1.
Published in final edited form as: Brain Res. 2020 Apr 21;1740:146850. doi: 10.1016/j.brainres.2020.146850

Table 2.

Small-molecule and genetic manipulations of the CRF system in alcohol use disorder.

Target Manipulation Result Reference
CRF Genetic KO KO mice consumed more alcohol than WT (Olive et al., 2002)
CRF1 receptor Genetic KO KO mice did not exhibit increase in alcohol self-administration during withdrawal (Chu et al., 2007)
KO mice exhibited decrease in HPA recruitment, measured by corticosterone (Pastor et al., 2008)
KO mice exhibited increase in alcohol intake and increase in withdrawal-included alcohol self-administration (Molander et al., 2012)
CRF2 receptor Genetic KO Alcohol intake was slightly reduced (Sharpe et al., 2005)
When stressed, KO animals consumed more alcohol than WT (Sillaber et al., 2002)
Alcohol intake was slightly reduced, but reduction was not maintained throughout the study (Kaur et al., 2012)
CRF1/CRF2 receptor Genetic KO KO mice did not exhibit alcohol-induced locomotor sensitization but exhibited decrease in HPA axis recruitment, measured by corticosterone in response to alcohol intake (Pastor et al., 2008)
Repeated forced swim increased alcohol intake (Pastor et al., 2011)
CRF-BP Genetic KO No change in alcohol intake in mouse DID paradigm (Ketchesin and Seasholtz, 2015)
CRF Genetic overexpression KO mice consumed less alcohol than WT (Palmer et al., 2004)
CRF1/CRF2 receptor Agonist Decrease in alcohol intake during two-bottle choice (i.c.v.) (Bell et al., 1998)
Decrease in alcohol intake in dependent vapor-exposed rats (i.c.v.) (Thorsell et al., 2005)
Increase in withdrawal-induced stress, measured by social interaction (i.c.v., intra-CeA, intra-BNST) (Huang et al., 2010)
Alcohol-withdrawn animals spent less time on the open arms of the elevated plus maze, indicating increase in the stress response (i.c.v.) (Zhao et al., 2013)
Decrease in social interaction as a measure of withdrawal-induced anxiety (i.c.v.) (Overstreet et al., 2004b)
Induction of alcohol reinstatement (i.c.v., intra-MRN) (Le et al., 2002)
CRF1/CRF2 receptor Antagonist (D-Phe CRF12/41) Decrease in anxiety-like behavior (i.c.v.) (Valdez et al., 2003)
Decrease in footshock-induced reinstatement (i.c.v.) (Liu and Weiss, 2003)
Decrease in alcohol-seeking behavior in dependent rats (intra-CeA) (Funk et al., 2006)
Decrease in alcohol withdrawal-induced alcohol self-administration (intra-CeA) (Finn et al., 2007)
Lower elevation of brain reward thresholds during alcohol withdrawal (i.c.v.) (Bruijnzeel et al., 2010)
CRF1 receptor Antagonist (MTIP) Decrease in anxiety-like behavior in elevated plus maze during acute alcohol withdrawal (i.p.) (Gehlert et al., 2007)
CRF2 receptor Agonist (Ucn3) Decrease in alcohol intake after escalation (i.c.v.) (Sharpe and Phillips, 2009)
(Valdez et al., 2004)
Increase in drinking in control animals (intra-CeA) (Funk et al., 2007)
CRF2 receptor Antagonist (astressin-2B) Decrease in alcohol intake (intra-VTA) No effect (intra-CeA) (Albrechet-Souza et al., 2015)
CRF-BP Antagonist (CRF[6–33])
CRF1/CRF2 receptor Antagonist (α-helical CRF) Decrease in anxiety-like behavior in elevated plus maze during acute alcohol withdrawal (i.c.v.) (Baldwin et al., 1991)
No change in anxiogenic-like effect of acute alcohol withdrawal (i.c.v.) (Rassnick et al., 1993)
Increase in alcohol preference in low-alcohol-preferring mice but not high-alcohol-preferring mice (i.c.v.) (O'Callaghan et al., 2005)
Decrease in alcohol intake in DID paradigm (i.c.v.) (Lowery and Thiele, 2010)
Decrease in anxiety-like behavior in elevated plus maze during acute alcohol withdrawal (intra-CeA) (Rassnick et al., 1993)
CRF1 receptor Antagonist (LWH-63) Decrease in withdrawal-induced alcohol self-administration (Sabino et al., 2006)
Decreases in alcohol intake and blood alcohol levels (s.c.) (Lowery-Gionta et al., 2012)
CRF1 receptor Antagonist (antalarmin) Decrease in acquisition of alcohol drinking and alcohol consumption (i.p.) (Lodge and Lawrence, 2003)
Decrease in alcohol self-administration and blockade of footshock-induced reinstatement in Marchigian Sardinian alcohol-preferring rats (i.p.) (Hansson et al., 2006)
Decrease in withdrawal-induced alcohol self-administration (i.p.) (Chu et al., 2007)
Decrease in withdrawal-induced alcohol self-administration in dependent but not nondependent rats (i.p.) (Funk et al., 2007)
Decrease in stress (yohimbine)-induced alcohol self-administration (i.p.) (Marinelli et al., 2007)
(Yang et al., 2008)
(Ayanwuyi et al., 2013)
CRF1 receptor Antagonist (CP-154,526) Decrease in alcohol withdrawal-induced stress, measured by social interaction (i.p.) (Overstreet et al., 2004b)
(Overstreet et al., 2004a)
(Breese et al., 2005)
(Wills et al., 2009)
Decrease in swim stress-induced alcohol intake in mice (i.p.) (Lowery et al., 2008)
CRF1 receptor Antagonist (CP-37,395) Decrease in alcohol intake (i.p.) (Giardino and Ryabinin, 2013)
(Simms et al., 2014)
CRF1 receptor Antagonist (CRA-1000) Decrease in alcohol withdrawal-induced stress, measured by social interaction (i.p.) (Knapp et al., 2004)
CRF1 receptor Antagonist (MJL-1–109-2) Decrease in withdrawal-induced alcohol self-administration in dependent but not nondependent rats (i.p.) (Funk et al., 2007)
CRF1 receptor Antagonist (MPZP) Decrease in withdrawal-induced alcohol self-administration in dependent but not nondependent rats (i.p.) (Gilpin et al., 2008)
Decrease in withdrawal-induced alcohol self-administration in dependent but not nondependent rats (s.c.) (Richardson et al., 2008)
CRF-BP Antagonist (CRF[6–33]) Decrease in alcohol intake (intra-VTA but not intra-CeA) (Albrechet-Souza et al., 2015)

BP, binding protein; KO, knockout; WT, wildtype; CeA, central nucleus of the amygdala; BNST, bed nucleus of the stria terminalis; VTA, ventral tegmental area; DID, drinking-in-the-dark; HPA, hypothalamic-pituitary-adrenal; MRN, median raphe nucleus.