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. 2021 Feb 4;15(1):262–272. doi: 10.1080/19336950.2021.1882112

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© 2021 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 5. — The molecular mechanism underlying the chronic inhibition of Ang II and PMA on I_Ks current in HEK 293B cell. (a) The representative I_Ks current by the Ang II and PMA treatment under the channel forward transport inhibition. (b) The current–voltage relationship for the I_Ks current by the Ang II and PMA treatment under the channel forward transport inhibition. (c) The summary data for the tail currents by the Ang II and PMA treatment under the channel forward transport inhibition at +40 mV prepulse. (d) The representative I_Ks current by the Ang II and PMA treatment under the channel endocytosis inhibition. (e) The current–voltage relationship for the I_Ks current by the Ang II and PMA treatment under the channel endocytosis inhibition. (f) The summary data for the I_Ks tail currents by the Ang II and PMA treatment under the channel endocytosis inhibition at +40 mV prepulse. (**P < 0.01, compared with the control group; ^##P < 0.01, compared with Brefeldin A or Dynasore group; ^#P < 0.05, compared with Brefeldin A group)