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. Author manuscript; available in PMC: 2021 Feb 9.
Published in final edited form as: Microb Pathog. 2018 Dec 5;127:212–219. doi: 10.1016/j.micpath.2018.12.008

Fig. 6. Mediators of innate immunity were downregulated in the absence of CD14.

Fig. 6.

The expression of CD14 in the lungs was significantly elevated in wild-type (WT) mice after each disease state, but not in the knockout (KO) mice (A). Tlr4 was significantly increased in the co-morbid WT mice but this gene was reduced in the KO (B). A marker of TH2 immunity, Retnla was increased in asthma and co-morbid conditions in the WT mice but not in the KO (C). Data are represented as the mean and SEM of n = 5 mice per group per genotype and analyzed by two-way ANOVA with Tukey’s multiple comparisons test. Data are significant (p < 0.05) when letters above bars are dissimilar.