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. 2020 Oct 26;476(2):699–713. doi: 10.1007/s11010-020-03937-x

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — CPEB4 was a target of miR-373-3p. a and b The mRNA and protein levels of CPEB4 in glioma and normal tissues, TMZ-resistant, and TMZ-sensitive glioma tissues were analyzed by qRT-PCR and western blot, respectively. c and d The mRNA and protein levels of CPEB4 in LN229/TR and SHG-44/TR cells compared to their parental cells (LN229 and SHG-44) were evaluated by qRT-PCR and western blot, respectively. e starBase v3.0 predicted there were miR-373-3p binding sites in the 3′UTR of CPEB4. f Dual-luciferase reporter assay was used in 293T cells to verify the correlation between CPEB4 and miR-373-3p. *P < 0.05