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. 2021 Feb 9;11:3426. doi: 10.1038/s41598-021-83022-5

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Cxcr7 is upregulated concomitantly with ERK activation in the border zone of myocardial infarction. (a) Cxcr7 mRNA levels in infarcted murine hearts. The heart tissue was dissected into two portions: a remote zone (RZ) and a border zone (BZ). Replicate samples were derived from heart tissues of wild-type mice (n = 7). Data are shown as paired data points. Significance was calculated by a paired t-test; **P < 0.01. (b) Immunoblot analysis of pERK and tERK in the RZ and BZ of infarcted heart. The bar chart shows the mean pERK-to-tERK ratio normalized to the value in the RZ. Samples were from infarcted heart tissues of Ctl mice (n = 6). Data are shown as paired data points. Significance was calculated by a paired t-test. *P < 0.05. (c) Immunoblot analysis of pERK and tERK in the BZ in infarcted hearts of Ctl and CKO mice. (d) Quantitative data of the results in Fig. 4c. Ctl, n = 6; CKO, n = 4. Data are shown as the mean ± SEM. Significance was calculated by an unpaired t-test. *P < 0.05.