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. 2021 Feb 9;14:30. doi: 10.1186/s13048-021-00775-3

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Propofol inhibits the progression of ovarian cancer via circVPS13C/miR-145 axis. a We transfected miR-145 or miR-NC into ovarian cancer cells, and the level of miR-145 was analyzed by qRT-PCR. (B-F) Ovarian cancer cells were divided into four groups, including vector + miR-NC group, Propofol + vector + miR-NC group, Propofol + oe-VPS13C + miR-NC group and Propofol + oe-VPS13C + miR-145 group. b Cell viability was assessed by MTT assay. c Cell cycle was measured by flow cytometry. d and e The migration and invasion capacities of ovarian cancer cells were assessed by transwell assays. f The apoptotic ovarian cancer cells were distinguished from normal or necrotic cells by flow cytometry. *P < 0.05, **P < 0.01, ***P < 0.001