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. 2020 Dec 31;11(2):413–422. doi: 10.1002/2211-5463.13061

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© 2020 The Authors. FEBS Open Bio published by John Wiley & Sons Ltd on behalf of Federation of European BiochemicalSocieties

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Fig. 4 — The impacts of miR‐505 inhibitor, si‐DLGAP1‐AS2+miR‐505 inhibitor and si‐DLGAP1‐AS2+si‐GALNT10 on malignant phenotype of HUCCT1 and RBE cells. (A,B) CCK‐8 assay for viability of HUCCT1 and RBE cells. **P < 0.01 versus NC group. (C,D) Colony formation assay for proliferation of HUCCT1 and RBE cells. **P < 0.01 versus NC group. (E,F) Transwell assay for migration and invasion of HUCCT1 and RBE cells. **P < 0.01 versus NC group. Scale bar: 200 µm. Error bar represents the mean ± SD derived from three independent experiments. Comparisons between groups were analyzed using t‐tests (two‐sided). OD, absorbance.