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. 2021 Jan 13;8(1):ENEURO.0011-20.2020. doi: 10.1523/ENEURO.0011-20.2020

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Copyright © 2021 Kadam et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution 4.0 International license, which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.

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Figure 5. — The srx-97 mutant phenotype is suppressed by other signaling mutants that appear to function downstream of SRX-97. A, Chemotaxis indices with respect to high concentration of benzaldehyde in WT, srx-97, osm-9, tax-2, tax-4, and gpc-1 mutants along with analysis of each mutant in the srx-97 background. B, Chemotaxis indices with respect to high concentration of benzaldehyde in WT, srx-97, odr-3, and srx-97; odr-3 mutants. This graph also indicates chemotaxis indices with respect to benzaldehyde on ablation of the AWC neuron [AWC(–)] in WT, srx-97, and odr-3 mutants. The assays were performed in triplicate over multiple days. Each dot in both plots A, B indicates an assay done in triplicate. The numbers at the base of each graph in A, B indicate the total number of times the experiment was performed with each genotype. The error bars represent SEM, and statistical significance is represented as “ns” for not significant; *p < 0.05, **p < 0.01, ***p < 0.001.