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. 2021 Jan 14;11:570867. doi: 10.3389/fphar.2020.570867

TABLE 2.

Anti-MIRI effects of saponins from Aralia.

Compound Major plant source Geographical distribution of plants Dose/Concentration Models Mechanism
Total saponins of Aralia elata (Miq) Seem Aralia elata (Miq) Seem Northeast China, North Korea, Japan, Russia 25, 50, 100 mg/kg, i.g. Rat MIRI model (in vivo) (Wang R. et al., 2018) Alleviating calcium homeostasis imbalance and endoplasmic reticulum stress-related apoptosis
30, 60 mg/kg; 1.25, 2.5 and 5 mg/ml Dog hemodynamic indexes (in vivo), Ca2+ transients and sarcomere shortening detection (in vitro) (Wang et al., 2014) Positive inotropic effect by maintenance of calcium homeostasis and increase of PKCε-dependent signaling pathway
Total saponins of Aralia taibaiensis Aralia taibaiensis Midwest China 60, 120, 240 mg/kg, i.g.; 25, 50 μg/ml Rat MIRI model (in vivo); H9c2 cardiomyocytes HR model (in vitro) (Yan et al., 2015) Suppressing apoptosis via the AMPK pathway
Elatoside C Aralia elata (Miq) Seem Northeast China, North Korea, Japan, Russia 2, 10, 50 nM Rat global MIRI injury model (ex vivo) (Wang M. et al., 2015) Attenuating oxidative stress and calcium overload through the activation PI3K/Akt and ERK1/2 and JAK2/STAT3 signaling pathway and inhibiting the opening of mPTPs
12.5, 25, 50 μM H9c2 cardiomyocytes HR model (in vitro) (Wang et al., 2014) Activating STAT3 signaling pathway and reducing ER stress-associated apoptosis
Araloside C Aralia elata (Miq) Seem Northeast China, North Korea, Japan, Russia 0.5, 1, 2.5 μM; 3.125, 6.25, 12.5, 25 μM Rat global MIRI injury model (ex vivo); H9c2 cardiomyocytes HR model (in vitro) (Wang et al., 2017; Du et al., 2018) Reducing oxidative stress, ER stress and calcium overload by regulating Hsp90
6.25, 12.5, 25 μM H2O2-induced H9c2 cardiomyocyte injury (in vitro) (Wang et al., 2019) Reducing oxidative stress by regulating mitochondrial function and AMPK activation
Calenduloside E Aralia elata (Miq) Seem Northeast China, North Korea, Japan, Russia 0.02, 0.1, 0.5 μg/ml H2O2-induced H9c2 cardiomyocyte injury (in vitro) (Tian et al., 2017) Inhibiting oxidative stress, apoptosis and calcium overload