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. 2021 Jan 12;11:598353. doi: 10.3389/fphar.2020.598353

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Copyright © 2021 Lin, Lin, Yang, Lu, Sun, Sun, Meng, Chi and Guo.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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SGLT1 influenced apoptosis in DCM via the JNK and p38 pathway. (A–B) Expression of p-p38, p-Erk and p-JNK in rats (n = 10). *p < 0.05 compared with the control group; ^# p < 0.05 compared with the DCM group. Experiments were performed in triplicate. (C–H) Western blot of p-p38, p-Erk and p-JNK in H9C2 cells. *p < 0.05 compared with the control group; ^# p < 0.05 compared with HG group. Experiments were performed in triplicate.