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. 2021 Feb 15;11:3828. doi: 10.1038/s41598-021-83270-5

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Changes in Cerebral Hemodynamics during Asphyxia and CPR: The changes in cerebral oxy-hemoglobin concentration (Δ[HbO₂]; first row), deoxy-hemoglobin concentration (Δ[Hb]; second row), total hemoglobin concentration (ΔTHC; third row) and tissue oxygen saturation (ΔStO₂; bottom row) from baseline during asphyxia (left), and from 1-min of CPR during CPR (right), are summarized and grouped by CPR outcome (ROSC, blue; No ROSC, red). Data are plotted as median (thick solid line) and interquartile range (IQR; thin solid line). The optimal decision threshold for ROSC are also plotted (dotted line) for Δ[HbO₂]_CPR (+ 1.3 µmol/L), ΔTHC_CPR (+ 3.4 µmol/L), and ΔStO_2,CPR (+ 2.1%). In the 10th minute of CPR, association with ROSC was assessed by Wilcoxon Rank-Sum test (**, p < 0.01; ***, p < 0.001).