Table 2.

The dysregulation and function of five overlapping DEmiRNAs (miR-101-3p, miR-143-3p, miR-145 (3p/5p), miR-34c-5p, and miR-146a-5p) in the osteodifferentiation of BMSCs and DPSCs, respectively.

	BMSC osteodifferentiation	DPSC osteodifferentiation
miR-101-3p	The overexpression of miR-101 promoted the osteogenic differentiation of BMSCs by targeting EZH2/Wnt/β-catenin signaling [34].	No evidence.
miR-143-3p	The downregulation of miR-143 promoted the osteogenic differentiation of BMSCs by being competitively combined with lncRNA MALAT1 and upregulating Osterix (Osx) [35].	The downregulation of miR-143 promoted the osteogenic differentiation of DPSCs by activating the NF-κB [36] and OPG/RANKL signaling [37].
miR-145 (3p/5p)	The downregulation of miR-145 promoted the osteogenic differentiation of BMSCs by targeting semaphorin3A (SEMA3A) [38].	The downregulation of miR-145 promoted the odontoblast differentiation of DPSCs by targeting KLF4 and OSX [39].
miR-34c-5p	The upregulation of miR-34c inhibited osteoblast differentiation of BMSCs by targeting the Notch signaling [40].	No evidence.
miR-146a-5p	The overexpression of miR-146a inhibited the osteogenic ability of BMSCs by targeting Smad4 gene [41].	The overexpression of miR-146a-5p promoted osteodifferentiation of DPSCs by suppressing Notch signaling [42].