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. 2021 Feb 17;7(8):eabb6596. doi: 10.1126/sciadv.abb6596

Fig. 2. RLN gene delivery suppressed tumor growth and modified TME in KPC pancreatic tumor model.

Fig. 2

(A) The dosing schedule for the administration of pRLN. (B) Tumor growth monitored by IVIS imaging after different treatments and (C) the quantification of the imaging data (n = 4). (D) Tumor weight recorded on day 26 after the sacrifice of mice (n = 4). (E) Survival curves in each treatment group (n = 5). (F) Modification of the TME detected by IF staining and Masson Trichrome and the quantification of α-SMA, collagen expression, and CD3+ T cell infiltration (n = 5 samples per group). (G) Expression of α-SMA and collagen I detected with Western blot after the administration of PBS or pRLN (n = 3). (H) Relative expression of MMP2, MMP9, MMP13, and IFNγ detected by RT-PCR (n = 4). (I) Immune cell infiltration in the tumor tissue detected by flow cytometry (n = 4). Statistical significance was calculated in (C) using two-way analysis of variance (ANOVA) with multiple comparisons, in (D) using one-way ANOVA with multiple comparisons, in (E) using log rank test, and in (F), (H), and (I) using t test. *P < 0.05, **P < 0.01, and ***P < 0.001; ns, not significant; Tregs, regulatory T cells.