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. 2021 Feb 18;12(2):191. doi: 10.1038/s41419-021-03466-5

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A H&E staining revealed IR triggered tubular injury. B TUNEL assay revealed the apoptosis cells induced by IRI in mice kidney tissues. Red arrow indicates coagulative necrosis with tubular structures; black arrow indicates renal tubule vacuole; blue arrow indicates renal tubular necrosis and detachment. C RT-qPCR revealed expression of MEG3 over a 12-h post IRI in renal cortex of mice compared with that in the sham group. D RT-qPCR revealed the expression of MEG3 in HK-2 cells 6 h following IRI. **p < 0.01.