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Proposed molecular mechanism underlying the effects of PB2 on LPS-induced inhibition of cell viability and apoptosis in HUVECs. PB2, procyanidin B2; LPS, lipopolysaccharide; Cyt-c, cytochrome c; Apaf1, apoptotic peptidase activating factor 1; HUVEC, human umbilical vein endothelial cell.
