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. 2021 Feb 19;12(2):203. doi: 10.1038/s41419-021-03492-3

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A Immunoblot analysis of GBP5 in human GBM patient samples and normal brain tissues. GAPDH was used as loading control. B Immunoblot analysis of GBP5 in human glioma cell lines, immortalized normal astrocyte NHA, and immortalized primary human fetal astrocyte IPHFA cells. GAPDH was used as loading control. C Cancer Genome Atlas (TCGA) dataset analysis of the GBP5 mRNA expression levels in GBM subtypes and normal brain tissues. **p < 0.01; ***p < 0.001. Normal: n = 9; Neural (NE): n = 27; classical (CL): n = 80; mesenchymal (ME): n = 96. D TCGA dataset analysis of the relationship between the expression levels of GBP5 and the prognosis of GBM patients. p = 0.015. n = 331.