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. 2021 Feb 19;12(2):203. doi: 10.1038/s41419-021-03492-3

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — A Immunoblot analysis of GBP5 in U87-C1, U87-GBP5, U251-C1, U251-GBP5, SNB19shGFP, and SNB19shGBP5 cells. GAPDH was used as loading control. B Effect of GBP5 knockdown or overexpression on GBM cell proliferation. **p < 0.01; ***p < 0.001. n = 3. C Effect of GBP5 on the cell migration assessed by wound healing assay in 24 h. Cell migration index = migrated distance (at 24 h)/scratching distance (at 0 h). *p < 0.05; **p < 0.01. n = 3. D Effect of GBP5 on the cell invasion by the matrigel transwell assay. Invasion index = OD570 of invaded cells in treatment groups/OD570 of invaded cells in control groups. **p < 0.01; ***p < 0.001. n = 3.