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. 2021 Feb 9;11:620159. doi: 10.3389/fphar.2020.620159

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Copyright © 2021 Liu, Yin, Li, Jiang, Zhang, Zhang and Li.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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ST inhibited the activation of NLRP3 inflammasome and attenuated inflammation in SHRs (n = 8). (A) Western blot analysis of NLRP3 and pro-caspase-1 in WKY, SHR, SHR + ST groups. The data were presented in fold of control. (B) PCR analysis of the expression of NLRP3 and caspase-1 in above three groups. (C) ELISA analysis of the levels of IL-1β and IL-18 in above three groups. (D) Western blot analysis of NF-κB (p65) in above three groups. The data were presented in fold of control. All data were shown as mean ± standard deviation (SD) (error bars) from eight independent biological repeats experiments, each of which included three technical repeats. ^* p < 0.05; ^** p < 0.01.