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. 2021 Feb 24;12(2):211. doi: 10.1038/s41419-021-03493-2

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© The Author(s) 2021, corrected publication 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — Rats were infected with ov-HECTD3 or sh-HECTD3 AAV8. The rats in the sham group were infected with or without AAV8 NC served as the negative control. After the specimen collection, the following analysises were performed. A, B ALT and AST activities in perfusate. Data are mean ± SD, n.s., not significant; *P < 0.05 and **P < 0.01 by one-way ANOVA followed by Tukey’s test. n = 5 per group. C Histological damage in liver of rats. H&E staining was performed on paraffin-embedded section of rat liver tissues. Scale bar = 100 μm. D Histological scores were analyzed according to Suzuki’s criteria. Data are mean ± SD, n.s., not significant; **P < 0.01 by one-way ANOVA followed by Tukey’s test. n = 5 per group. E–G Liver tissues expression levels of IL-1β, IL-6, and IL-10 were analyzed with the relative ELISA kits. Data are mean ± SD, n.s., not significant; **P < 0.01 by one-way ANOVA followed by Tukey’s test. n = 5 per group.