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. 2021 Feb 22;45(4):38. doi: 10.3892/or.2021.7989

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Copyright: © Chen et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 8. — CK inhibits the growth of RCC cells in tumour xenografts. BALB/c mice were ectopically implanted with RCC cells, and when the tumour size reached ~100 mm³, the mice were injected with CK (intraperitoneal) every 3 days for 30 days. (A) Effects of CK on tumour volume. (B) Effects of CK on mouse body weight. (C) H&E staining and immunohistochemical analysis of changes in Ki-67 expression in the harvested tumours after different treatments as indicated. Representative images are shown (magnification, ×40). (D) Western blotting analysis of cleaved caspase-3 expression in tumours collected from different groups. *P<0.05 and **P<0.01 vs. control group. CK, ginsenoside compound K; RCC, renal cell carcinoma; H&E, haematoxylin and eosin.