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. 2020 Dec 19;13(3):3588–3604. doi: 10.18632/aging.202305

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Copyright: © 2020 Gao et al.

This is an open access article distributed under the terms of the Creative Commons Attribution License (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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The downregulation of circFASTKD1 benefits vascular endothelial cells under hypoxic conditions. (A, B) qRT-PCR was used to determine circFASTKD1 levels in HUVECs (A) and HCMECs (B) under hypoxic conditions. (C, D) CCK-8 assays were used to determine the growth curves of HUVECs (C) and HCMECs (D) with up/downregulated circFASTKD1 under hypoxic conditions. (E) Photographs from the tube formation, cell migration and wound healing assays in HUVECs with different treatments under hypoxic conditions. (F–H) Quantitative analyses of the tube formation (F), cell migration (G) and wound healing (H) assays in HUVECs under hypoxic conditions.