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. 2021 Feb 26;15:15. doi: 10.1186/s40246-021-00314-7

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — CXCL16 modulated cell viability of human neonatal cardiomyocytes treated with H/R. a, b After si-CXCL16#1 and si-CXCL16#2 transfections, CXCL16 expression was measured at both mRNA and protein levels using qRT-PCR and western blot assays, n = 3 independent experiments. **P < 0.01 vs. si-con. c, d qRT-PCR and western blot assays revealed the expression of CXCL16 after the transfection of pcDNA3.1-CXCL16, n = 3 independent experiments. **P < 0.01 vs. vector. e, f Cell viability was measured using CCK-8 analysis, n = 3 independent experiments. **P < 0.01 vs. control, ^##P < 0.01 vs. H/R. All data were exhibited as mean ± standard deviation (SD)