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. 2021 Feb 27;28:7. doi: 10.1186/s40709-021-00135-8

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — CircCTNNA1 knockdown inhibited CRC tumorigenesis in vivo. SW620 cells transfected with sh-con or sh-circCTNNA1 were injected into nude mice. Tumor volume (a) and tumor weight (b) in each group were determined. c The expression levels of circCTNNA1, miR-363-3p and CXCL5 in the tumor tissues of each group were detected using qRT-PCR. d The protein expression of CXCL5 in the tumor tissues of each group was measured using WB analysis. *p < 0.05, ***p < 0.001