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. 2020 Dec 29;180(1):136–147. doi: 10.1093/toxsci/kfaa184

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© The Author(s) 2020. Published by Oxford University Press on behalf of the Society of Toxicology.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 1. — No evidence for altered peptidome by DNCB modulation of the proteome: A and B, Upset plots (Conway et al., 2017) of intersections of distinct HLA-I peptides observed in 3 biological replicates of control and DNCB treated HaCaT and HaCaT HLA-A2 cells. The total number of distinct 8 − 15mer peptides observed for each condition are shown on the side bars. C and F, Cell component gene ontology classification of distinct genes from which peptidome originates using PANTHER (Mi et al., 2019) from 3 biological replicates of control and DNCB treated HaCaT and HaCaT HLA-A2 cells.