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. Author manuscript; available in PMC: 2021 Mar 1.
Published in final edited form as: Epilepsia. 2020 Sep 17;61(10):2301–2312. doi: 10.1111/epi.16670

Figure 2. There was no increase of proinflammatory cytokines TNF, IL-1β and IL-6 in Gabrg2+/− knockout, a mouse model of infrequent absence epilepsy.

Figure 2.

A-C. The brains from 6–8 months old Gabrg2+/− mice were dissected and processed for measurement of pro-inflammatory cytokines. Equal amounts of protein lysates (30μg) from each brain region were determined for cytokines including tumor-necrosis factor alpha (TNF) (A), interleukin 1β (IL-1β) (B) and IL-6 (C) with enzyme-linked immunosorbent assay (ELISA). The measurements in the het were normalized to the same brain region of their own wildtype littermates. Data were presented as mean ± S.E.M. N=5 mice for each group.