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SENP3 was involved in high glucose-induced endothelial–monocyte adhesion and related adhesion molecules expression: (a) high glucose-induced endothelial–monocyte adhesion was reversed by SENP3 knockdown, (b) SENP3 was involved in high glucose-induced ICAM-1 expression, and (c) SENP3 was involved in high glucose-induced VCAM-1 expression.
*p < 0.05 versus normal glucose, #p < 0.05 versus high glucose + control siRNA.
