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. 2021 Feb 25;15:843–855. doi: 10.2147/DDDT.S293382

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© 2021 Chen et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

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Pretreatment with breviscapine attenuated CME-induced myocardial apoptosis (n = 4 for each group). (A) The mRNA expression levels of Bcl-2, Bax and cleaved caspase-3 in cardiac tissues. (B) The protein expression levels of Bcl-2, Bax and cleaved caspase-3 in cardiac tissues. (C) Representative immunohistochemical pictures of Bcl-2, Bax and cleaved caspase-3 in cardiac tissues (×200 magnification; bar = 100 μm). Data are presented as the mean±standard deviation (SD). *P < 0.05 versus the sham group; ^#P < 0.05 versus the CME group; ^&P < 0.05 versus the CME + BE group.

Abbreviations: CME, coronary microembolization; BE, breviscapine; LY, LY294002.