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The senescence induction phenotype is hypothesized to be a result of conflicting cues from cellular damage stimuli and expansion stimuli. In the context of this study, the expansion stimulus was provided by 10% FBS (equine model) and 10% FBS + bFGF + TGF-β1 (human model), while the cellular damage stimulus was provided by 10 Gy of irradiation. Figure created with BioRender.com.
