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. 2021 Mar 2;18:64. doi: 10.1186/s12974-021-02115-0

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Increased secretion of CCL2 after spinal cord injury (SCI). a The levels of CCL2 was significantly increased at 12 h and 1 day after SCI compared with that of sham group (n = 3 mice/per group). b QRT-PCR quantitative analysis of CCL2 expression at 12 h and 1 day after SCI and sham (n = 8 mice/per group). c The protein level of CCL2 was obviously higher in SCI group than that of sham group at 1 day after surgery (n = 3 mice/per group). *P < 0.05; **P < 0.01; ****P < 0.0001

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