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. 2021 Feb 10;14:627846. doi: 10.3389/fncel.2020.627846

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Copyright © 2021 Wang, Lei, Zhao, Xu, Dong, Ran, Shi and Fu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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GB inhibited the post-synaptic excitability of hippocampal neurons exposed to hypoxia in vivo. (A) Representative traces showing spontaneous discharges reflecting spontaneous excitatory post-synaptic potentials (sEPSCs) recorded from hippocampal slices obtained from the control, GB, hypoxia and hypoxia+GB groups. (B,C) Summary results for the effects of GB on sEPSCs. Data in (C) are expressed as the mean ± SEM (n = 9). **P < 0.01 vs. control group; ^#P < 0.05 vs. hypoxia group (Student's t-test).