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. 2021 Feb 18;8:618574. doi: 10.3389/fcell.2020.618574

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Copyright © 2021 Cheng, Wang, Wu, Cui, Wei, Liu and Wang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The depletion of circPostn relieves myocardial injury in MI mice. (A–D) The left ventricular chamber of 3-day post-MI mice was injected with the lentiviral vector of control shRNA or circPostn shRNA. (A) The expression of circPostn was measured by qPCR in mice. (B) The myocardium injury was analyzed by hematoxylin and eosin (H&E) staining. (C) The infarct size was assessed. (D) The LVEF, LVFS, LVAWd, and LVPWd were measured by echocardiography analysis. The ratio of heart weight (HW)/body weight (BW) and left ventricle weight (LVW)/BW was calculated. Data are presented as mean ± SD. Statistically significant differences were indicated: **p < 0.01, ^##p < 0.01.