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. 2021 Feb 21;11(2):563. doi: 10.3390/ani11020563

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© 2021 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Loading plots describing relationships between: (a) mammary gland relative expression of 91 genes (as detailed in Table S1) and biopsy collection time point, (b) gene functions: heat shock (HS), prolactin signalling (PRLSig), oestrogen signalling (ESig), cell proliferation (PROL), autophagy (AUT), apoptosis (APOP), morphology (MOR), and immunity (IMM), and (c) in vivo and in vitro treatments from a principal component analysis. Figure 3c included genes averaged heat shock (HS), prolactin signalling (PRLSig), oestrogen signalling (ESig), cell proliferation (PROL), autophagy (AUT), apoptosis (APOP), morphology (MOR), and immunity (IMM), by in vivo and in vitro treatments CL-H, CL-C, CL-B, HT-H, HT-C, and HT-B.