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. 2021 Mar 4;11:5241. doi: 10.1038/s41598-021-84812-7

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Echocardiographic evaluation of cardiac function and remodelling in a mouse model of myocardial infarction. (a) Schematic study outline. (b) Ejection fraction (EF) at the time of inclusion in week 0. (c) EF after 4 weeks of dosing with either vehicle or captopril. (d) EF change over study period (week 4-week 0)/week 4*100%. (e) Left ventricle internal diameter in diastole (LVIDd) at the time of inclusion in week 0. (f) LVIDd at week 4. (g) LVIDd change over study period. (h) Left ventricle anterior wall dimension/thickness (LVAW) in diastole and (i) systole. (j) Left ventricle posterior wall dimension/thickness (LVPW) in diastole and (k) systole. Data is presented as mean ± s.e.m. n = 10–12. One-way ANOVA with Tukey’s post hoc test. Significance: *p < 0.05, **p < 0.01, ***p < 0.001. #: Significant (p < 0.05) after removal of single non-responder in LAD—Captopril. LAD: left anterior descending artery ligation.