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. 2021 Mar 4;14:82. doi: 10.1186/s13104-021-05489-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Association of DEG identified in NEC samples and IL-17 target genes. IL-17 receptor (IL-17R) can bind to IL-17A, IL-17F or IL-17A/F to activate various signaling pathways leading to the regulation of IL-17 targets genes involved in tissue remodeling and anti-microbial activity as well as mediators of inflammation as adapted from [15, 20]. Genes identified as being significantly modulated in NEC samples compared with control non-NEC samples are labeled in green and red while those for which no significant differences were notes are labeled in grey