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. 2021 Mar 5;16(3):e0248168. doi: 10.1371/journal.pone.0248168

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© 2021 Chang et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 3 — Microarray data (GSE120103) from the GEO databank was utilized for clinical association study. (A) HOTAIR levels in lesions from severe endometriosis patients (stage 4) (Endo; n = 18) were analyzed and compared with that in normal endometria (Normal; n = 18). (B) Expression levels of component genes in HOTAIR-associated complexes, PRC2 and LSD1/REST/CoREST, were also analyzed and compared with the levels in healthy controls. (C) Expression levels of possible downstream effectors of HOTAIR, including genes at the HOXD locus and other validated targets [17,28], were compared between patients (Endo) and controls (Normal) by using t-test.