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. 2021 Mar 6;32(3):26. doi: 10.1007/s10856-021-06499-6

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Temporal regulation of soluble VE-cadherin from endothelialized gene-free scaffold and SDF-1α GAS. SDF-1α GAS strongly affects the vascular growth of endothelial cells by suppressing the release of soluble VE-cadherin. Coculturing with ADSCs offers further control on the release of soluble VE-cadherin from endothelial cells. HUVECs on SDF-1α GAS demonstrated significant reduction in the levels of soluble VE-cadherin at days 7 (p < 0.05) and 10 (p < 0.0005) relative to HUVECs on gene-free scaffold. Coculture on SDF-1α GAS strongly attenuated the release of VE-cadherin at all time points. Data are presented as mean ± standard deviation. One-way ANOVA was used to deduce statistical significance. *, **, ***, and **** indicate statistical significance of p < 0.05, p < 0.01, p < 0.005, and p < 0.0005, respectively