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Silencing Malat1 in CMECs aggravated oxidative stress, apoptosis and mitochondrial dysfunction after myocardial infarction (MI).
MDA content, total SOD activities, GSH and GSSG levels, and ROS and H2O2 generation in the border zone (A). Relative expression of mitochondrial apoptosis-related genes and mitochondrial fusion- and fission-related genes (B). *p < 0.05 compared with the sham group, #p < 0.05 compared with the MI + Scr-shRNA group. n = 6 in each group
