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. 2021 Jan 12;476(4):1751–1763. doi: 10.1007/s11010-020-04020-1

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Regulation of LLC-WRC 256 cell proliferation and apoptosis by circSlc7a11. a Alterations of circSlc7a11 expression in LLC-WRC 256 cells by transfection with overexpressing vectors or siRNAs. The expression of circSlc7a11 in cells was detected using quantitative RT-PCR. b Positive regulation of LLC-WRC 256 cell proliferation by circSlc7a11. The proliferation rates of LLC-WRC 256 cells with altered circSlc7a11 expression were measured using an MTS assay. c Inhibition of LLC-WRC 256 cell apoptosis by circSlc7a11. The percentage of apoptotic LLC-WRC 256 cells was determined by flow cytometry. Slc7a11: solute carrier family 7 member 11 cystine–glutamate transporter; NC: negative control; OD490: absorbance at 490 nm; **P < 0.01; ***P < 0.001; ***P < 0.0001