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. 2021 Feb;9(3):228. doi: 10.21037/atm-20-4431

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2021 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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Effects of BCCAO, EMS, and URB597 on cortical and hippocampal neovascularization. (A) Immunopositivity to the endothelial cell marker CD34, indicative of neovascularization (green), was found at the cortical-temporal muscle interface following EMS surgery (red line) (magnification: 200×); (B) compared with the BCCAO group, the number of CD-34-positive microvessels (MVNs) was increased in the surgical area of cortex in EMS group rats (magnification: 200×). (C,D) Histogram showing MVN for each group. EMS but not URB957 enhanced MVN. ^#P<0.05 vs. BCCAO; ^▲P<0.05 vs. BCCAO+URB. (C: F_4,35 =4.287, P<0.05; D: F_3,28 =2.713, P>0.05). EMS, encephalomyosynangiosis; BCCAO, bilateral common carotid artery occlusion.