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. 2021 Mar 9;23:78. doi: 10.1186/s13075-020-02373-y

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Overexpression of miR-503-5p results in promoted proliferation and reduced apoptosis of rat primary chondrocytes. a The expression efficiency of miR-503-5p verified by RT-qPCR assay. b Apoptosis of rat primary chondrocytes after overexpression or downregulation of miR-503-5p detected by flow cytometry. c The expression of Caspase-3, Bax, and Bcl2 in primary chondrocytes after overexpression or downregulation of miR-503-5p detected by Western blot analysis. d Statistics of c. e Proliferation of rat primary chondrocytes after overexpression or downregulation of miR-503-5p detected by MTT assay. f The expression of inflammatory cytokines in rat primary chondrocytes examined by ELISA. *p < 0.05. Measurement data were expressed as mean ± standard deviation. Comparisons among multiple groups were conducted by one-way ANOVA with Tukey’s post hoc test. The experiment was repeated three times