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. 2021 Mar 11;12(3):256. doi: 10.1038/s41419-021-03548-4

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Green and red spots indicate the genomic location of 5’ and 3’ FISH probes for the FGFR2 gene. Distinct orange and green signals in more than 20% of the tumor cells represent FGFR2 rearrangement. Schematic representation of FGFR2 gene translocation (a) and non-translocation (b). Overall survival curve (c) and disease-free survival curve (d) stratified by FGFR2 translocation and non-translocation cases in ICCs (Kaplan–Meier method). The radiologic, gross and histopathologic (20× and 200×) images of three representative patients whose tumor possessed an FGFR2 translocation (e). Three ICCs, which respectively numbered No. 201336830, No. F201501983, and No. 201505169 showed small, large, and multiple tumors, in radiologic or gross morphology, were all mass-forming (MF) type. For histological classification, these were all pancreaticobiliary type adenocarcinoma as presented in 20× and 200× HE staining.