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. 2021 Feb 25;11:628359. doi: 10.3389/fonc.2021.628359

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Copyright © 2021 Qiu, Zheng and Meng

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Summary of the cellular metabolic effects of hyperglycemia in cancer. GLUT1, Glucose transporter 1; PPP, Pentose phosphate pathway; EGFR, Epidermal growth factor receptor; IF, Inflammatory factors; TF, Transcription factors; HIF, Hypoxia-inducible factor-1; HK2, Hexokinase2; PKM1, Pyruvate kinase M1; EMT, Epithelial-mesenchymal transition; FASN/FAS, Fatty acid synthase; ER, Estrogen receptor; IGF, Insulin growth factor; PI3K, Intracellular phosphatidylinositol kinase; AKT, Protein kinase B; mTOR, Mammalian target of rapamycin; Grb2, Growth factor receptor-bound protein2; MAPK, Mitogen-activated protein kinase.