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. 2021 Mar 12;6(3):E87–E93. doi: 10.1055/a-1371-9359

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The Author(s). This is an open access article published by Thieme under the terms of the Creative Commons Attribution-NonDerivative-NonCommercial-License, permitting copying and reproduction so long as the original work is given appropriate credit. Contents may not be used for commercial purposes, or adapted, remixed, transformed or built upon. (https://creativecommons.org/licenses/by-nc-nd/4.0/).

This is an open-access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives License, which permits unrestricted reproduction and distribution, for non-commercial purposes only; and use and reproduction, but not distribution, of adapted material for non-commercial purposes only, provided the original work is properly cited.

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Fig. 1 — A 20-year-old male patient with acute left lower abdominal pain in the previous three days and a normal CRP value (<5 mg/l). a B-mode US image shows an echogenic, non-compressible lesion adjacent to the air-filled sigmoid colon. b Illustration of the manifestation of EA in the B-mode US image in image A. Arrowheads indicate the echogenic, non-compressible lesion as a typical B-mode US pattern of EA. The lesion is adjacent to the air-filled sigmoid colon (*). c CEUS after 33 s shows inhomogeneous enhancement with a small central area of non-enhancement, surrounded by hyperenhancement of the inflammatory fatty tissue. d Illustration of the manifestation of EA on the CEUS image in image C. Arrowheads indicate the hyperenhanced inflammatory fatty tissue. The centrally located infarcted fat tissue shows non-enhancement (*). The air-filled sigmoid colon is bordered in red.