Figure 6. Connexin 40 (Cx40) overexpression increases vascular relaxation and decreases right ventricular systolic pressure (RVSP) in hypoxia‐exposed mice.
A, Cx40 protein level in mouse pulmonary endothelial cells (ECs) from normoxia‐exposed control adenovirus (Cont‐Adv)–injected mouse (Nor+Cont‐Adv), hypoxia‐exposed Cont‐Adv–injected mouse (Hypo+Cont‐Adv), and hypoxia‐exposed Cx40‐Adv–injected mouse (Hypo+Cx40‐Adv). Dot plot shows summarized data. Nmice=5 per group. B, Vascular response in small distal (fourth‐fifth order) pulmonary arteries. Nor+Cont‐Adv, nmice=10; Hypo+Cont‐Adv, nmice=11; Hypo+Cx40‐Adv, nmice=10. Left: EC‐dependent relaxation. Middle: Endothelium‐dependent hyperpolarization (EDH)–mediated relaxation. Right: Smooth muscle (SM)–dependent relaxation. C, Typical record of right ventricular pressure (RVP) and summarized data of RVSP. Nor+Cont‐Adv, nmice=7; Hypo+Cont‐Adv, nmice=7; Hypo+Cx40‐Adv, nmice=5. D, Fulton Index (right ventricle [RV]/(left ventricle [LV]+septum [S])). Nor+Cont‐Adv, nmice=5; Hypo+Cont‐Adv, nmice=5; Hypo+Cx40‐Adv, nmice=4. Data are mean±SEM. *P<0.05 vs Nor+Cont‐Adv. # P<0.05 vs Hypo+Cont‐Adv. Statistical comparison of dose‐response curves among 3 groups was made by 2‐way ANOVA with Bonferroni post hoc test. Statistical comparison of RVSP among 3 groups was made by 1‐way ANOVA with Bonferroni post hoc test. ACh indicates acetylcholine; and SNP, sodium nitroprusside.