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. 2021 Mar 14;20:25. doi: 10.1186/s12940-021-00718-x

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — The sex-specific interaction analysis on PM_2.5, SIRT1_391 and mortality. Note: The male participants without two SIRT1_391 minor allele copies and with low exposure of PM_2.5 was regarded as the reference group. The model adjusted for age at baseline, sex, education, marriage, occupation, residence, exercise, smoking, and alcohol consumption