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. 2021 Mar 3;22(5):2519. doi: 10.3390/ijms22052519

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© 2021 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Molecular targets of pharmacological conditioning. GPCR = G protein-coupled receptor; TNF-R2 = tumor necrosis factor receptor 2; gp130 = glycoprotein 130; TRPV4 = transient receptor potential vanilloid 4; VEGFR-1 = vascular endothelial growth factor receptor 1; SAFE = survivor activating factor enhancement; TNFα = tumor necrosis factor alpha; JAK/STAT = janus kinase/signal transducers and activators of transcription; RISK = reperfusion injury salvage kinase; PI3K = phosphatidylinositol 3-kinase; PKB = protein kinase B; ERK = extracellular-signal-regulated kinase; GSK3β = glycogen synthase kinase 3 beta; NO/PKG = nitric oxide/protein kinase G; eNOS = endothelial nitric oxide synthase; cGMP = cyclic guanosine monophosphate; miRNA = micro ribonucleic acid; mBK_Ca = mitochondrial large-conductance calcium-sensitive potassium channel, mK_ATP = mitochondrial adenosine triphosphate-sensitive potassium channel; mPTP = mitochondrial permeability transition pore.